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SO TELL ME, HOW DOES THIS WORK?

 The first step in understanding how OXYRICH does what it does for a person is to appreciate the unique bio-electrical manufacturing process.

Put simply, purified water (cleansed of all organic compounds and impurities) with a little salt added (to facilitate an electrolyte solution) is exposed to a serious electric charge. This process of electrolysis progressively "de-composes" the water by removing hydrogen from the H20 and by uniquely bonding the surplus oxygen molecules together, enabling the solution to remain stable whilst "super concentrating" the oxygen level.

This procedure increases the oxygen from that which is found in tap water (about 8 parts per million) and multiplied up to the OXYRICH solution of 50,000 parts per million (5% of total volume). The oxygen is "stabilised" (meaning 'locked in' to solution and prevented from becoming a 'gas') Samples from the OXYRICH Production Facility have been shown by independent laboratories to remain stable for at least two years in the unopened container and eight months in the opened container.This represents a world first breakthrough not only due to the stable qualities of OXYRICH but due to the pH of 7.9 to 8.2 which makes it safe to drink!
Older technologies require a chemical process that results in potentially toxic pH of 2.4 (H₂O₂) or pH of 12.5 (chemical salts products)
which means only a few drops are safe to ingest.  

Typically atmospheric oxygen (a gas) is delivered to the body via the lungs. In contrast, Oxyrich (although rich in oxygen) is not a gas, it is an aqueous solution and like all solutions (eg water, alcohol etc) is absorbed via the gastro-intestinal tract, across the gastric membrane.  

As the gastric membrane is similar in nature to the tissues of the lungs it is probable that oxygen 'locked in solution' crosses over into the blood stream in the same way and this by-passes the first two steps of aerobic respiration (ventilation / gas exchange).  

The oxygen in a 10ml dose of Oxyrich whilst not in itself a lot of oxygen (1.5ml) compared to normal respiratory oxygen is however in a very concentrated form (15%) in the solution.  

Research conducted in Japan, USA, UK and Australia has shown that following the use of (between 5% and 15% versions) OXYRICH and dosages of 6-15ml, marked increases in the partial pressure of arterial oxygen (POZ), of around 5%-8% have been measured. 

In addition, exercise testing at Victoria University in Australia and the University of Hull in the UK during 2005 have shown an increase of between 3 and 6% increase in oxygen uptake at the an-aerobic threshold. This increase of usable oxygen would reduce times by 3 minutes per hour for an elite tri-athlete (cycle leg) and is in the order of 150-200ml per minute increased oxygen uptake. Given that the OXYRICH dose of 10ml would only account for 1.5ml of OZ, the enhanced performance must be due to a secondary mechanism.  

HOW CAN THIS BE HAPPENING? Exercise physiologists have been aware for a long time that a phenomenon known as "de-saturation" exists among some athletes where the haemoglobin 'dumps' oxygen into the muscles, thus increasing the oxygen available to the muscles without increasing cardio-respiratory rates. A similar event may be encouraged by taking OXYRICH prior to the intense exercise event. This could explain the disproportionate OZ metabolism (150-200mllmin) from ingesting only 1.5ml from a 10ml dose of OXYRICH.  

POSSIBLE MECHANISMS? 

Observers of oxygen therapy, using traditional forms of O2 in solution (hydrogen peroxide) even in very small doses (5 drops) have recorded changes to enzyme 2,3 DPG, in the haemoglobin this can lead to a shift in the 'HbO²' dissociation curve to the left and by speeding up the delivery of oxygen to the muscle tissues, improve the efficiency of existing haemoglobin and cardio-respiratory functions.  

This is being conducted at Victoria and Monash Universities to investigate the possible link between SPORTS OXYSHOT (Oxyrich), Enzyme 2,3 DPG and the enhanced performance reported by elite athletes and observed in the laboratory trials in the UK, USA and Australia.

 

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